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心室晚电位是发生于QRS波终末部或ST段上的低振幅、高频率的电信号,这些信号可能由于传导延缓的病变心肌所产生,并可作为致心律失常的信号。急性心肌梗塞(AMI)1～4周内,若出现晚电位,预示以后6～24月发生心源性猝死与室性快速心律失常的危险性增高。近年,已确认在 AMI 数小时内,应用溶栓治疗是一种增加存活及维护心肌功能的有效方法。然而,关于早期 AMI时,晚电位的发生率、过程及溶栓治疗的资料则很少。故本文对 AMI 初10天时,溶栓治疗对晚电位的发生率和演变进行了评价。
The ventricular late potentials are low-amplitude, high-frequency electrical signals that occur at the terminal or ST segment of QRS. These signals may be produced by the diseased myocardium that conducts delay and serve as a signal for arrhythmia. Acute myocardial infarction (AMI) 1 to 4 weeks, if there is late potential, indicating that after 6 to 24 months of sudden cardiac death and ventricular tachycardia risk increased. In recent years, it has been confirmed that thrombolytic therapy is an effective method to increase survival and maintain myocardial function within a few hours of AMI. However, with regard to early AMI, the incidence of late potential, the process and the thrombolytic therapy is very little information. Therefore, this article on the first 10 days of AMI, thrombolytic therapy on the incidence and evolution of late potentials were evaluated.