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作者对肝硬变合并有食道静脉曲张者与慢性肝炎各5例作对比观察,发现肝硬变合并门脉高压症的胃粘膜特点是1.血管内皮细胞增生,基膜增厚;2.粘膜层水肿;3.粘膜下层动静脉分流增多;4.淋巴间隙增宽等。与Sarfeh氏等对大鼠所作的实验研究结果相似。这提示肝硬变门脉高压胃粘膜病变的主要原因在于门脉压力增高本身而非其它原因。本文所测两种疾病的胃泌素未见有统计学的差异。
The author of cirrhosis combined with esophageal varices and chronic hepatitis in each of 5 cases for comparison and found that liver cirrhosis with portal hypertension is characterized by 1. Vascular endothelial cell proliferation, thickening of the basement membrane; 2. Mucosal Stratum edema; 3. submucosal arteriovenous shunt increased; 4. lymphatic space widened. Similar to Sarfeh et al.’s experimental study in rats. This suggests that the main cause of portal hypertension in patients with gastric mucosal lesions is increased portal pressure itself rather than other reasons. There was no statistical difference in gastrin between the two diseases tested in this study.