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为了证明光化学反应导致脑血栓形成时心肌是否有形态学改变，给麻醉大鼠静注Ｒｏｓｅｂｅｎｇａｌ（７．５ｍｇ／ｋｇ）后，用５６０ｎｍ的绿光照射暴露的颅骨，诱导脑血栓形成，探讨血栓性脑缺血时心肌超微结构的改变并作线粒体体视学分析。结果表明，脑血栓形成后心肌形态学改变明显，其组织病理学特征既有别于缺血性坏死又不同于缺血再灌注所致的心肌损伤．脑血栓形成动物的心肌线粒体体视学参数：平均体积、体密度、面数密度及数密度等显示，与内膜有关的参数多为可逆改变，而与外膜相关的参数则不然．是否因脑血栓形成时心肌肌浆网和线粒体ＡＴＰ酶活性抑制所致，尚待研究．
In order to prove whether there is morphological changes in myocardium during photothrombosis caused by cerebral thrombosis, rats were anesthetized with intravenous injection of Rosebengal (7.5mg / kg), exposed to 560nm green light exposure to the exposed skull to induce thrombosis Changes of myocardial ultrastructure during cerebral ischemia and mitochondrial stereological analysis. The results showed that the morphological changes of myocardium after cerebral thrombosis were obvious. The histopathological features were different from those of ischemic necrosis and myocardial ischemia-reperfusion. Myocardial mitochondrial stereological parameters of cerebral thrombosis animals: mean volume, body density, number density and number density showed that most of the parameters related to intima were reversibly changed, while the parameters related to adventitia were not. Is due to inhibition of myocardial sarcoplasmic reticulum and mitochondrial ATPase activity in the formation of cerebral thrombosis, yet to be studied.