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有报告认为甲状旁腺激素(PTH)过多,是人们长期寻找的引起尿毒症异常的毒素之一。尿毒症病人,由于其甲状旁腺增生,分泌增多,PTH的肾清除及降解减少,血中的PTH常升高,Massary收集材料表明这种激素是引起神经病学异常以及搔痒、骨坏死、尿毒症性高脂血症和阳萎的尿毒症毒素之一。Avram及其同事根据透析的病人其平均红细胞压积显著增加,网状细胞增多,及根据Better等的观察,证实在甲状旁腺次全切除后,骨髓的钙化恢复,因而推测PTH升高可加重尿毒症的贫血。作者回顾性分析了42例和前瞻性观察了17例维持血透的尿毒症病人,研究其运动神经传导速度(MNCV)与血浆PTH浓度的关系,以探讨PTH过
There are reports that excessive parathyroid hormone (PTH), is one of the long-sought looking toxins that cause abnormalities in uremia. Uremic patients, due to hyperplasia and secretion of parathyroid glands, increased renal clearance and degradation of PTH, PTH levels in the blood are often elevated, Massary collected material shows that this hormone is caused by neurological abnormalities and itching, osteonecrosis, uremia Hyperlipidemia and impotence of uremic toxins. According to Avram and colleagues, hemodialysis patients showed a significant increase in mean hematocrit and increased reticulocytes, and according to Better et al., Confirmed restoration of calcification in the bone marrow following subtotal parathyroidectomy and speculated that elevated PTH may be exacerbated Uremia of anemia. The authors retrospectively reviewed 42 patients and prospectively observed 17 cases of hemodialysis patients with uremia to study the relationship between motor nerve conduction velocity (MNCV) and plasma PTH concentration to explore the PTH