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尽管成人呼吸窘迫综合征(ARDS)的病因错综复杂,但病理变化较为一致。其主要病理变化是肺充血、出血、微血栓、水肿以及肺泡萎缩,数天后形成透明膜。病程较长时,肺组织常常出现炎症病灶,后期水肿吸收,表现为间质纤维化和肺气肿。电镜下超微结构变化是毛细血管充血和微血栓形成,肺泡隔水肿,含有较多的细胞成分,肺Ⅰ型上皮细胞和内皮细胞肿胀,空泡形成或破坏,Ⅱ型细
Although the causes of adult respiratory distress syndrome (ARDS) are complex, the pathological changes are more consistent. The main pathological changes are pulmonary congestion, hemorrhage, microthrombus, edema and alveolar contraction, a few days after the formation of a transparent membrane. Long course of the disease, the inflammation of the lung tissue often appear in the late absorption of edema, manifested as interstitial fibrosis and emphysema. Electron microscopic changes in ultrastructure are capillary congestion and microthrombus formation, alveolar septum, with more cellular components, lung type I epithelial cells and endothelial cell swelling, vacuolization or destruction, type II thin