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本组54例,男30例,女24例。年龄32～78岁,平均62岁。尿量均少于平常。少尿(400ml/日)者21例,占38.89%。尿常规检查:尿蛋白:微量7例,(+)5例,(++)9例,(+++)1例共22例占40.74%。尿渣镜检有红细胞者6例占11.11%,有白细胞(>5个/高倍镜视野)者5例占9.26%,管型3例占5.56%。尿素氮:测定30例占55.56%。大于7mmol/L 的7例占23.33%。非蛋白氮测定25例,大于25mmol/L 5例占20%。肌酐测定24例,大于177mmol/L 5例占20.83%。讨论本组肺心病急性心力哀竭病人肾功能发生变化,其机理主要是:(1)肺心病急性心哀时,静脉压增高,肾脏淤血。(2)缺氧、二氧化碳潴留引起肾小球滤过膜通透性增高。(3)外周小动脉收缩,包
The group of 54 patients, 30 males and 24 females. Age 32 to 78 years old, average 62 years old. Urine volume less than usual. Oliguria (400ml / day) in 21 cases, accounting for 38.89%. Urine routine examination: urinary protein: trace 7 cases, (+) 5 cases, (++) 9 cases, (+++) 1 case a total of 22 cases accounted for 40.74%. Urine residue were detected in 11.11% of 6 cases of erythrocytes, white blood cells (> 5 / high magnification) 5 cases accounted for 9.26%, 3 cases accounted for 5.56%. Urea nitrogen: 30 cases accounted for 55.56%. Seven cases of more than 7mmol / L accounted for 23.33%. Non-protein nitrogen determination in 25 cases, more than 25mmol / L 5 cases accounted for 20%. Creatinine in 24 cases, more than 177mmol / L 5 cases accounted for 20.83%. Discussion of this group of patients with acute cardial heart failure pulmonary heart disease renal function changes, the main mechanism is: (1) pulmonary heart disease acute heart failure, increased venous pressure, kidney congestion. (2) hypoxia, carbon dioxide retention caused glomerular filtration membrane permeability increased. (3) peripheral arterioles shrink, package