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本实验用SD大鼠复制冷束缚应激性损伤动物模型,观察次黄嘌呤—黄嘌呤氧化酶体系(A组)和别嘌呤醇(B组)对应激时发生的急性全身适应综合征的影响。结果显示在应激性组织损伤和体内脂质过氧化反应方面,A组比B组严重(P<0.05),证明黄嘌呤氧化酶来源的自由基参与了大鼠的冷束缚应激性损伤过程。
In this experiment, SD rats were used to replicate animal models of cold-impaired stress injury, and the effects of hypoxanthine-xanthine oxidase system (group A) and allopurinol (group B) on acute systemic adaptation syndrome occurred during stress were observed. . The results showed that in the stress tissue injury and lipid peroxidation reaction in vivo, group A was more severe than group B (P<0.05), demonstrating that xanthine oxidase-derived free radicals are involved in cold-binding stress injury in rats. .