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随着内源性的气体分子一氧化氮(NO)和内源性一氧化碳(CO)的发现,将低氧性肺动脉高压(HPH)发病机制的研究带入了新的阶段。以往的研究提示,NO和CO在低氧性肺血管结构重塑中发挥
With the discovery of endogenous nitric oxide (NO) and endogenous carbon monoxide (CO), the study of the pathogenesis of hypoxic pulmonary hypertension (HPH) has been brought to a new stage. Previous studies have suggested that NO and CO play a role in hypoxic pulmonary vascular remodeling