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病例:男,60岁。有高血压病史13年,平素血压21.3/12.5kPa左右,最高23.9/14.6kPa,近一年来未用降压药物。两天前晨超打拳时,意外被人推擅(未摔倒),出现头部发胀与轻微头疼,当日下午头疼加重,伴恶心、四肢无力,当时测血压为22.6/13.3kPa,自服心痛定10mg,头疼稍有减轻后入睡,次日头疼进一步加剧,测血压26.6/18.6kPa,门诊以高血压病收住心血管内科,经治疗后血压降至正常,但头疼无减轻。于入院第3天行头颅CT检查,发现左侧尾状核头部及同侧侧脑室前角为高密度影,出血量约3毫升,随后转入神经内科。体检:两眼底视乳头边界清,视网膜动脉变细。反光增强,
Case: Male, 60 years old. Hypertensive history of 13 years, usually about 21.3 / 12.5kPa blood pressure, the highest 23.9 / 14.6kPa, nearly a year without antihypertensive drugs. Two days ago when the morning boxing, the accident was pushed good (did not fall), there was a slight headache and headache, the afternoon increased headache, with nausea, weakness, blood pressure was 22.6 / 13.3kPa, self-service Heartache given 10mg, headache slightly reduced to sleep, headache further intensified the next day, measuring blood pressure 26.6 / 18.6kPa, outpatient admitted to cardiovascular medicine with hypertension, after treatment, blood pressure dropped to normal, but without any headache. On the third day after admission, head CT examination revealed that the anterior horn of the left caudate nucleus and the ipsilateral lateral ventricle was a high-density shadow with a blood loss of about 3 ml and was subsequently transferred to the neurology department. Physical examination: the two end of the nipple clear boundary, retinal artery thinner. Reflective enhancement,