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在心绞疼发病机理上起主要作用的,有两种因素——解剖和血管神经的结合影响。冠状血循环之机能一解剖的障碍,导致急性心肌缺氧和心绞疼的发作。心肌物质代谢的障碍,和因而在心肌那里引起疼痛性的物质(BoΛeBыxBeⅢecTB)的形成,是心疼急速产生的基础。基本上可以认为,被我们利用于治疗心绞疼的菸草酸,能改善工作中的心肌的化学过程,和比较相当长时间地减少急性发作或者介除患者于疼综合症。
In the pathogenesis of angina pectoris plays a major role, there are two factors - the combination of anatomy and vascular nerves. The function of the coronary blood circulation is an anatomical obstacle that leads to the onset of acute myocardial hypoxia and angina pectoris. Obstacles to myocardial metabolism and the formation of painful substances in the myocardium (BoΛeBыxBeⅢecTB) form the basis for the rapid onset of distress. Basically, the tobacco acids we use to treat angina pectoris can be thought of as improving the chemical processes in the working myocardium and reducing acute attacks or mediating pain syndromes for a relatively long time.