论文部分内容阅读
钾(K~+)、钙(Ca~(++))、钠(Na~+)和镁(Mg~(++))在实验性心律失常的发生中,均参与作用。然而,在临床上,绝大多数的这种心律失常是由于钾浓度的变化所引起。因为在临床所遇到的疾病的各种电解质波动水平范围内,K~+是最易改变心脏电生理性质的电解质。因此,本文主要讨论K~+浓度失常所起的作用,并简短地涉及其他电解质的可能作用。钾钾和经膜动作电位(tranmembraneaction potential) 本节将简短地论及K~+在(1)维持经膜动作电位(AP),(2)激动传导,和(3)激动形成中的作用。在舒张期时(动作电位的第4位相),细
Potassium (K ~ +), calcium (Ca ~ (++)), sodium (Na ~ +) and magnesium (Mg ~ (++)) are all involved in the development of experimental arrhythmia. However, in clinical practice, the vast majority of these arrhythmias are caused by changes in potassium concentration. Because of the range of electrolyte imbalances encountered in clinical conditions, K ~ + is the most accessible electrolyte that changes the electrophysiological properties of the heart. Therefore, this article focuses on the role of K ~ + concentration disorders and briefly addresses the possible role of other electrolytes. Potassium Potassium and Transmembrane Action Potential This section will briefly address the role of K ~ + in (1) maintaining the transmembrane action potential (AP), (2) agonistic conduction, and (3) activation. In the diastolic (action potential of the fourth phase), fine