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Stein和Leventhal曾报道过一组继发闭经的妇女,其双侧卵巢是增大的多囊卵巢。虽然这些病人半数有多毛症,但尿中17-酮类固醇仍在正常范围内。Stein提出在此综合症中应除外“肾上腺皮质紊乱”。在多囊卵巢的病因学探讨中,大多数讨论是围绕着下丘脑的作用或卵巢的作用而展开的。有的发现血清促黄体生成激素(LH)是增高的,另一些则发现血清LH高低不一。在多囊卵巢切除的楔形部份和卵巢静脉血中发现有过量的雄激素,联想到某些病人的失调是卵巢的酶发生紊乱,而另一些病人则是由于来自肾上腺的雄激素过量而引起的。有些病人则表现为肾上腺和卵巢的雄激素都异常。本研究试图对36例确诊为多囊卵巢综合症的LH分泌失常进行探索。
Stein and Leventhal have reported a group of women with secondary amenorrhea whose bilateral ovaries are enlarged polycystic ovary. Although half of these patients have hirsutism, urinary 17-ketosteroids are still in the normal range. Stein proposed that “adrenocortical disorders” should be excluded in this syndrome. In the etiology of polycystic ovary, most discussions focus on the role of the hypothalamus or ovarian function. Some found that serum luteinizing hormone (LH) is elevated, while others found that serum LH levels vary. In polycystic ovary resection of the wedge and ovarian venous blood found in excess androgen, reminiscent of some patients with ovarian enzymes is disorders, while others are due to adrenal androgen caused by an overdose of. Some patients show abnormalities in the adrenal and ovarian androgens. This study attempted to explore the diagnosis of LH in 36 cases of polycystic ovary syndrome.