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采用大鼠蛛网膜下隙出血模型,经光镜和Vidas图像分析的方法,对不同时间段的脑动脉平滑肌的横断面积(S1),内膜的横断面积(S2),脑动脉壁横断面积(S3),脑动脉腔横断面积(S4),V1:平滑肌的体密度(S1/S3);V2:内膜的体密度(S2/S3);D1:蛛网膜下降面脑动脉壁厚度;D2:脑面脑动脉壁厚度进行了观察测定,发现蛛网膜下隙出血后脑血管痉挛具有双相性,管腔缩窄,管壁增厚,急性脑血管痉挛比迟发性脑血管痉挛缩窄更明显,蛛网膜面管壁增厚比脑面管壁增厚明显,大脑中动脉的变化比基底动脉变化更明显,实验各组管壁各成分的体密度没有显著差异,说明脑动脉痉挛并不是由于管壁成分增加引起.
Using the model of subarachnoid hemorrhage in rats, the cross-sectional area (S1), the intimal area (S2), the cross-sectional area of the cerebral artery wall S3), the cross-sectional area of the cerebral artery lumen (S4), V1: the body density of the smooth muscle (S1 / S3); V2: the intima density (S2 / S3) Cerebral artery wall thickness was observed and found that subarachnoid hemorrhage after cerebral vasospasm with biphasic, lumen narrowing, wall thickening, acute cerebral vasospasm than delayed cerebral vasospasm constriction is more obvious, The thickening of the arachnoid surface wall was more obvious than the thickening of the brain surface. The change of the middle cerebral artery was more obvious than that of the basilar artery. There was no significant difference in the body density of the various components of the wall of the experimental group, indicating that cerebral artery spasm was not caused by the tube Wall composition caused by increased.