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近年来,有人提出由屯积于肺的白血细胞为主组成的微聚物对肺的损伤起了很重要的作用。我们已肯定在失血性休克过程有肺损伤出现是以RES抑制为前提的。此过程中,白血细胞有何作用是值得探讨的。本文以“肺扣押”、“肝释放”和肺结构改变为指标,观察正常时、单纯失血性休克、单纯RES封闭和RES封闭后休克过程的白血细胞动态变化及其和肺损伤的关系。用胶性碳封闭RES可引起循环白细胞数降至封闭前的39％,有显著统计差异(P<0.0001)。在封闭后休克10’和30’见到白血细胞由肝释放(P<0.001和0.005),在肺扣押(P
In recent years, it has been suggested that the polymer mainly composed of white blood cells accumulated in the lung plays a very important role in lung injury. We have confirmed that the occurrence of lung injury during hemorrhagic shock is based on the inhibition of RES. In this process, what is the role of white blood cells is worth exploring. In this paper, “lung seizure”, “liver release” and lung structure changes as an indicator to observe the normal, simple hemorrhagic shock, RES closure and RES shock after closure of white blood cells and its dynamic changes in the relationship between lung injury. Closing RES with colloidal carbon resulted in a significant statistical difference (P <0.0001) in circulating leukocyte counts down to 39% before occlusion. Shock white blood cells were released from the liver at 10 ’and 30’ post-closure shock (P <0.001 and 0.005)