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吸入NO和服用磷酸二酯酶5(PDE-5)抑制剂对于急性肺损伤引起的肺动脉高压患者的肺血管有选择性扩张作用。若给予患者磷酸二酯酶-5抑制剂,应用到足以降低患者肺动脉压的剂量,已被证明会降低动脉的氧合。为研究在单独应用时不会降低肺动脉压,又可以促进急性肺损伤动物模型对吸入NO
Inhalation of NO and selective administration of phosphodiesterase 5 (PDE-5) inhibitors to pulmonary vasoconstriction in patients with pulmonary hypertension caused by acute lung injury. Administration of a phosphodiesterase-5 inhibitor to patients at doses sufficient to reduce pulmonary arterial pressure has been shown to reduce arterial oxygenation. In order to study the application will not reduce pulmonary artery pressure alone, but also can promote acute lung injury animal model of inhaled NO