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过去曾把恢复期额外的氧耗称为“氧债”.近几年的研究证明,运动后的过量氧耗(EPOC)是由于运动导致体内稳态被扰乱,如运动引起的组织温度升高,细胞内外离子的变化,以及代谢和激素水平的变化等,这些生理变化持续地存在于运动后的恢复期内.因此,乳酸并非形成氧债的原因,而只是作为恢复过程的燃料,其氧化并不会导致额外氧耗.
In the past, extra oxygen consumption during recovery was referred to as “oxygen debt.” In recent years, studies have shown that post-exercise excess oxygen consumption (EPOC) is due to movement leading to disturbance of homeostasis, such as exercise-induced tissue temperature Elevated, changes in intracellular and extracellular ions, as well as changes in metabolism and hormone levels, these physiological changes persist in the recovery period after exercise.Therefore, lactic acid is not the reason for the formation of oxygen debt, but only as a recovery process of fuel, Its oxidation does not result in additional oxygen consumption.