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吉兰—巴雷综合征(GBS)常继发于细菌和病毒感染。本文报告一例环状孢子(cvclospora)感染后发生GBS,并假设环状孢子是GBS的另一感染因子。 患者为58岁男性,发病前一周发生腹泻及发热。入院当日,患者清醒、无力伴四肢感觉改变;18小时内发生四肢瘫,反射消失,给予机械通气。静注免疫球蛋白三日后,进行5个疗程的血浆置换治疗。第五天脑脊液检查示总蛋白60mg/dl,无细胞增多。开始治疗三天内,患者可移动下肢。病后25天出院,所有肌群均能对抗地心引力。出院三周内,患者可扶拐缓行,两个月后可勉强行走50码,但双手仍显著力弱。
Guillain-Barre syndrome (GBS) often secondary to bacterial and viral infections. This article reports a case of GBS following infection with cvclospora and assumes that circulatory spores are another infectious agent of GBS. The patient was a 58-year-old man with diarrhea and fever a week earlier. On the day of admission, the patient was awake, unable to feel changes in their extremities; quadriplegia occurred within 18 hours, the reflex disappeared, and mechanical ventilation was given. After intravenous immunoglobulin three days, 5 courses of plasma exchange treatment. Cerebrospinal fluid examination on the fifth day showed total protein 60mg / dl, no cell increase. Within three days of beginning treatment, the patient can move the lower extremity. 25 days after discharge, all muscle groups can fight against gravity. Within three weeks of discharge, the patient could be excused slowly and could barely walk 50 yards two months later, his hands were still significantly weaker.