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磁共振成像(MRI)尚未广泛用于研究一氧化碳(CO)中毒后的脑损害。作者报告3例4年前同时发生CO中毒患者的临床后果及脑MRI发现。 例1,男,43岁,被送入急诊室以10升/分非再呼吸式面罩(non—rebreathing mask)吸氧。血压120/80mmHg(16/10.7KPa),呼吸36次/分。呈嗜睡状,无局灶神经缺陷。ECG示右束支传导阻滞和非特异性ST—T波异常,血白细胞计数(WBC)为20800(20.8×10~9/L),肌酸激酶(CK)2293IU/L(正常<220),碳氧血红蛋白氢氧化钠试验阴性。持续面罩吸氧,几小时后完全清醒。既往无神经系统疾患史。2天后WBC12700,9天后CK50IU/1,ECG无改变。2周后脑CT左丘脑显示低密度灶。随后出现严重的抑郁症,住院并抗抑郁治疗。4年
Magnetic resonance imaging (MRI) has not been widely used to study brain damage after carbon monoxide (CO) poisoning. The authors report 3 patients with CO poisoning 4 years ago, the clinical consequences and brain MRI findings. Example 1, male, 43 years old, was admitted to the emergency room to receive oxygen at 10 l / min using a non-rebreathing mask. Blood pressure 120 / 80mmHg (16 / 10.7KPa), breathing 36 beats / min. Drowsiness, no focal nerve defects. ECG showed right bundle branch block and nonspecific ST-T wave abnormalities. The WBC was 20800 (20.8 × 10 9 / L), creatine kinase (CK) was 2293 IU / L (normal <220), carbon Oxyhemoglobin sodium hydroxide test negative. Continuous mask oxygen, fully awake after a few hours. No previous history of neurological disorders. After 2 days WBC12700, 9 days CK50IU / 1, ECG no change. After 2 weeks, the CT CT of the left thalamus showed low-density foci. Subsequent severe depression, hospitalization and antidepressant treatment. 4 years