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药物性肺水肿是非心源性肺水肿的一种。由于病因不同,故其病机也不完全相同;有些还属于复合因素或机理尚未阐明。药物性肺水肿多数系药物中毒所致,机理为肺毛细血管渗透性增高。国外报道较多的是由于海洛英、美沙酮、非那酮、丙醇酚、大仑丁、硝基呋喃、碳胺等药物中毒所致。上述药物可以引起中枢性呼吸功能紊乱、肺功能障碍、肺脏通气血流比值失衡、肺内静动脉血分流增多,肺顺应性减低,造成肺泡通气低下、肺泡活性物质缺损、严重低氧血症、酸中毒;这些
Drug-induced pulmonary edema is a kind of non-cardio pulmonary edema. Due to different causes, so its pathogenesis is not exactly the same; some still belong to the complex factors or mechanisms have not yet been elucidated. Most of the drug-induced pulmonary edema caused by drug poisoning, the mechanism for pulmonary capillary permeability increased. More foreign reports are due to heroin, methadone, non-ketone, propanol, Dalen Ding, nitrofuran, carbamine and other drug poisoning caused. These drugs can cause central respiratory dysfunction, pulmonary dysfunction, lung ventilation, blood flow ratio imbalance, pulmonary venous shunt increased, decreased lung compliance, resulting in low alveolar ventilation, alveolar reactive material defects, severe hypoxemia, Acidosis; these