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ＭＲＬ－Ｆａｓ’ｌｐｒ／小鼠能自发产生多系统器官非特异性自身免疫性疾病，被认为可作为研究自身免疫性内耳病的动物模型。其血管纹毛细血管有抗体沉积，却无炎性细胞浸润，抗体可能通过直接与细胞成分结合而干扰细胞功能，从而损害血管纹，不过耳蜗损害也可能由遗传或?..
MRL-Fas’lpr / mice spontaneously produce nonspecific autoimmune diseases of multiple system organs and is considered as an animal model for the study of autoimmune inner ear disease. The vascularized capillaries have antibody deposition but no inflammatory cell infiltration. Antibodies may interfere with cellular functions by binding directly to cellular components, thereby impairing the vascular pattern, although the damage to the cochlea may also be caused by genetic or.