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胸膜结核患者半数以上表现为胸膜的继发病变。许多患者具有结核肺实质的表现。由于缺乏简便诊断方法,诊断胸膜结核则较为困难,确诊需找到分枝杆菌。通常根据无免疫活性患者的PPD皮试阳性和胸膜活检到肉芽肿做出推测诊断,但在有免疫活性的患者或结核高发区,PPD皮试便失去其诊断价值。ADA是嘌呤代谢中催化腺苷和脱氧腺苷脱氨生成次黄苷和脱氧次黄苷的一种酶。ADA_1和ADA_2是两种同工酶。前者可为单体或二聚体,见于实质器官和淋巴细胞,后者见于血清和单核细胞。重症联合免疫缺陷患者缺乏ADA活性,故认为ADA与免疫反应有关。ADA增高
More than half of patients with pleural tuberculosis secondary to pleural lesions. Many patients have tuberculosis pulmonary parenchyma performance. Due to the lack of simple diagnostic methods, diagnosis of pleural tuberculosis is more difficult to confirm the need to find mycobacteria. PPG skin tests and pleural biopsies to granulomas are usually presumed to be speculative based on non-immunocompetent patients, but PPD skin tests lose their diagnostic value in immunocompetent patients or high TB areas. ADA is an enzyme that catalyzes the deamination of adenosine and deoxyadenosine to purify inosine and deoxyinosine in purine metabolism. ADA_1 and ADA_2 are two isozymes. The former can be monomeric or dimeric, found in parenchymal organs and lymphocytes, the latter seen in serum and monocytes. ADA activity in patients with severe combined immunodeficiency deficiency, ADA and therefore that the immune response. ADA increased