瑞舒伐他汀调节血管内皮生长因子促进动脉瘤栓塞后内皮祖细胞的动员

来源 :中国临床神经科学 | 被引量 : 0次 | 上传用户:a715362633
下载到本地 , 更方便阅读
声明 : 本文档内容版权归属内容提供方 , 如果您对本文有版权争议 , 可与客服联系进行内容授权或下架
论文部分内容阅读
目的观察大鼠腹主动脉瘤栓塞模型和内皮祖细胞(EPCs)培养中,瑞舒伐他汀对血管内皮生长因子(VEGF)和EPCs的促进和调节作用。方法选取雄性SD大鼠48只,随机分为2组:未用药组(n=24)和瑞舒伐他汀组(n=24)。通过血管移植端侧吻合方法构建腹主动脉瘤栓塞后模型,并经灌胃给予瑞舒伐他汀20 mg·kg-1·d-1治疗。采用流式细胞术检测药物治疗后外周血循环EPCs水平变化并通过ELISA评估外周血VEGF水平。瑞舒伐他汀组大鼠骨髓分离的EPCs,采用MTT方法检测不同剂量瑞舒伐他汀对培养的P2代EPCs活性的影响,划痕实验和成血管实验检测EPCs的功能变化水平;使用定量聚合酶链反应(q PCR)对EPCs分化相关的Ephrin B2和Eph B4表达量进行检测。结果动脉瘤栓塞造模后,瑞舒伐他汀可以在第10天明显提高外周血VEGF水平,并引起第30天时循环EPCs明显提高;经鉴定被0.001μmol·L-1瑞舒伐他汀干预18 h后,表现出明显的促进EPCs活性作用,并显著提升了EPCs的迁移和成管能力;同时q PCR检测表明Ephrin B2表达水平有明显提高。结论瑞舒伐他汀可提升外周血VEGF和EPCs水平,能促进EPCs的活性、迁移和成管能力。 Objective To observe the effects of rosuvastatin on vascular endothelial growth factor (VEGF) and EPCs in rat models of abdominal aortic aneurysm embolism and endothelial progenitor cells (EPCs). Methods Forty eight male Sprague Dawley rats were randomly divided into 2 groups: untreated group (n = 24) and rosuvastatin group (n = 24). The model of abdominal aortic aneurysm embolization was established by end-to-side anastomosis of vascular grafts and treated with rosuvastatin 20 mg · kg-1 · d-1 by intragastric administration. Flow cytometry was used to detect the level of EPCs in peripheral blood after drug treatment and the level of VEGF in peripheral blood was evaluated by ELISA. The EPCs isolated from the rat bone marrow of rosuvastatin group were assayed by MTT assay. The effects of different dosages of rosuvastatin on the activity of EPCs on passage P2 were detected. Scratch and angiogenesis assays were used to detect the changes of EPCs activity. Chain reaction (q PCR) was used to detect the expression of Ephrin B2 and Eph B4 in EPCs. Results Rosuvastatin could significantly increase the level of VEGF in peripheral blood on day 10 after induced by embolization of aortic aneurysm, and significantly increased the circulating EPCs on the 30th day. It was identified that rosuvastatin was inhibited by 0.001μmol·L-1 rosuvastatin for 18 hours Afterwards, EPCs showed obvious activity of promoting EPCs, and significantly enhanced the migration and tube formation ability of EPCs. At the same time, q PCR detection showed that the expression of Ephrin B2 was significantly increased. Conclusion Rosuvastatin can enhance the levels of VEGF and EPCs in peripheral blood and promote the activity, migration and tube formation of EPCs.
其他文献
糖尿病是一种代谢性疾病,是心血管疾病的独立危险因素.糖尿病心肌病是独立于冠状动脉疾病和高血压的糖尿病并发症之一,是一种多因素所致的复杂疾病,可导致较高的发病率及病死
人在睡眠阶段会出现独特的神经-体液活动及血流动力学的改变,然而睡眠障碍会引发和加重器质性或功能性心血管疾病,如高血压、心力衰竭和冠心病等。其中,阻塞性睡眠呼吸暂停低
糖尿病心肌病是糖尿病引起的一种特异性心肌病,它的发病机制非常复杂,其中内质网病变发挥了重要作用.内质网通过内质网应激及钙调蛋白维持心肌细胞功能的稳态,内质网应激一定
先天性心脏病(CHD)治疗技术的快速进展使得在发达国家有超过90%的CHD儿童能存活到成年,导致成人CHD患者数量远远超过儿童CHD患者数量.心律失常患者可表现为从无症状到有致命性
药物洗脱支架在显著降低再狭窄发生率的同时,增加了晚期支架内血栓发生风险及加速了新生内膜动脉粥样硬化.过去认为再狭窄的发生主要是由于新生内膜过度增殖,但新近研究发现,
心室重构是多种损伤因素作用于心脏,引起心脏的代谢、结构及功能改变,它不仅是一种心脏损伤的结果,也是引起心律失常、心力衰竭、猝死等心血管疾病的基础.而核因子-κB是一个
心力衰竭是心血管疾病的终末阶段,其相关生物标志物研究已受到广泛关注.随着对生长分化因子15病理生理学的研究,发现其与心力衰竭密切相关.现通过对生长分化因子15生物学特性
一、直接利用公式求解例1电路由电池A、B、C并联组成.电池A、B、C损坏的概率分别是0.3,0.2,0.2,求电路断电的概率.
本文结合各类设计规范,结合作者实践工作经验,对二级负荷电源的选择与配电设计做出了探讨。
目的研究脑动静脉畸形(AVMs)经立体定向放射治疗后不同时期的组织病理变化。方法对10例脑AVMs立体定向放射治疗后的病理标本进行苏木精-伊红染色,并用平滑肌肌动蛋白(SMA)、