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本文分析1965年～1985年治疗的慢性增生性下颌骨骨髓炎110例,男68例,女42例。小儿87例,15岁～18岁7例,成年16例。病程3月至15年.初期感染源是乳磨牙或恒磨牙根尖周炎和冠周炎。初期症状是中度肿胀,以后颌骨变形。在加重期疼痛剧烈,肿胀加剧,开日困难,体温升高,睡眠食欲不好。加重期症状消失以后,骨变形更明显,颔骨周围呈鞘状增厚,病变1年可加重10次～12次。如病变扩展到喙突和髁突,发生继发性关节炎和变形性关节病.病变区牙齿发生逆行性牙髓炎,成为继发感染源;恒牙胚感染或坏死也是继发性感染源。X 线检查,在初期牙根尖周骨组织吸收。很快发生骨膜反应,形成骨化性骨膜炎,增生的骨皮质有分层线,反应疾病的临床周期(缓解期和加重期),固有的
This article analyzes 110 cases of chronic hypertrophic mandibular osteomyelitis from 1965 to 1985, 68 males and 42 females. 87 cases of children, 15 years old to 18 years old in 7 cases, 16 cases of adult. Course of disease from March to 15. The initial source of infection is molars or molars periapical and pericoronitis. The initial symptoms are moderate swelling, after the deformation of the jaw. Exacerbation of severe pain, swelling, opening difficulties, body temperature, poor sleep appetite. Exacerbation of the symptoms disappeared later, the more obvious bone deformation, tarsal sheath sheath thickening lesions 1 year may increase 10 times to 12 times. Such as lesions extended to the coracoid process and condylar, secondary to arthritis and degenerative joint disease .Deformed lesions of the teeth of retrograde pulpitis, a secondary source of infection; permanent tooth germ infection or necrosis is also a secondary source of infection . X-ray examination, the initial apical bone tissue absorption. Periosteal reaction occurs soon, the formation of ossifying periostitis, hyperplastic cortical bone layering line, the clinical cycle of response to disease (remission and aggravation), the inherent