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60年代以来,美国及许多欧洲国家使用人绝经期促性腺激素(HMG)与柠檬酸氯芪醛胺(CC)作为卵巢刺激剂,因此在未来的几十年中发生的卵巢肿瘤可能较以前增多。HMG与CC均可直接或间接地使促性腺激素超过生理性循环浓度。本文报道1979～1992年期间于使用卵巢刺激时或其后发生的12例粒层细胞瘤患者。 例1为37岁妇女。停服12服避孕剂后闭经,左侧卵巢轻度肿大。腹腔镜与活检无恶性病变。使用CC与甲磺酸溴化麦角环肽1周期后无效,其后用4周期HMG亦未受孕。2年后患者有不规则出血及左卵巢再次肿大,剖腹行左侧卵巢切除,并证实
Since the 1960s, the United States and many European countries have used menopause gonadotropin (HMG) and citrate chlorcemidamine (CC) as ovarian stimulants, so that ovarian tumors may develop more in the coming decades than before . Both HMG and CC can directly or indirectly promote gonadotropin over physiological circulating concentrations. This article reports 12 patients with meningioma who developed during or after ovarian stimulation from 1979 to 1992. Example 1 is a 37-year-old woman. After taking 12 contraceptives amenorrhea, left ovarian slightly enlarged. Laparoscopy and biopsy no malignant lesions. CC and methanesulfonic acid bromocriptine using 1 cycle after the invalid, then use the 4-cycle HMG also not pregnant. 2 years after the patient had irregular bleeding and left ovarian re-enlargement, cesarean section left ovariectomy and confirmed