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正常动物的直接检查还没有提供关于阻止自身免疫初发机制的详细资料.应用一种间接但更有效的方法检查自身免疫过程的调节,有助于研究抑制已形成应答动物再次发生严重的自身免疫的机制。这方面的研究多属实验性变应性脑脊髓炎(EAE)。关于甲状腺自身免疫机制的了解尚少,有人提出调节机制对实验性变应性甲状腺炎(EAT)的削弱是否与EAE 相似,为了解对EAT 再次应答的调节因素,作者检查了已发生EAT大鼠对再次免疫的敏感性.根据实验目的不同,用不同方法诱导甲
Direct examination of normal animals has not provided detailed information on the mechanisms that prevent the initiation of autoimmunity.It is an indirect but more effective way to examine the regulation of autoimmune processes and to help study ways to suppress the recurrence of severe autoimmunity in established animals Mechanisms. Most of the research in this area is experimental allergic encephalomyelitis (EAE). There is little known about the autoimmune mechanism of the thyroid. It is suggested whether the attenuation of experimental allergic thyroiditis (EAT) by the regulatory mechanism is similar to that of EAE. To understand the regulatory factors that reoccur the EAT, the authors examined whether EAT rats Sensitivity to re-immunization. Depending on the purpose of the experiment, A is induced in different ways