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原发性开角型青光眼的发病机制与小梁和睫状肌部位细胞外基质的异常沉积密切相关,基质金属蛋白酶及其组织抑制剂通过调节细胞外基质的降解和代谢平衡参与了原发性开角型青光眼的发病机制。
The pathogenesis of primary open-angle glaucoma and trabecular and ciliary muscle parts of the extracellular matrix abnormal deposition is closely related to the matrix metalloproteinases and tissue inhibitors through the regulation of extracellular matrix degradation and metabolic balance involved in the primary Pathogenesis of open angle glaucoma.