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目的应用临床电生理手段对斜视性弱视的中枢及外周损害作一些探讨。方法使用丹麦Ｅｖｏｍａｔｉｃ４０００型电生理仪，全视野翻转棋盘格作刺激，选择９０’、４６．６’、２０．７’三种空间频率，Ｐ－ＥＲＧ与Ｐ－ＶＥＰ同步记录的方法，对２３例单眼斜视性弱视儿童进行电生理检测，弱视眼Ｐ—ＥＲＧ记录使用双眼注视、注视标调整寻找ｂ波最大振幅的方法，并与正常对照组儿童进行比较。结果弱视眼Ｐ－ＶＥＰＮ７５潜伏期、Ｐ１００潜伏期较正常儿童组延长，Ｐ１００振幅较正常儿童组降低。弱视眼Ｐ－ＥＲＧ与正常儿童组比较并无明显异常。结论斜视性弱视的损害、神经传导的时间延迟主要发生在视觉通路视网膜以上的部位，视网膜并无明显受损。
Objective To investigate the central and peripheral lesions of strabismic amblyopia by clinical electrophysiological methods. Methods The Evomatic 4000 electrocardiograph was used in Denmark. The total visual field was changed to checkerboard for stimulation. Three spatial frequencies of 90 ’, 46.6’ and 20.7 ’were selected. P-ERG and P-VEP were simultaneously recorded. Twenty-three Single strabismus amblyopia children electrophysiological examination, amblyopia P-ERG recording using binocular gaze, gaze adjustment to find the maximum amplitude of b wave method, and compared with the normal control group of children. Results The latency of P-VEPN75 and the latency of P100 in amblyopic eyes were longer than those in normal children. The amplitude of P100 was lower than that in normal children. There was no significant difference between P-ERG and normal children in amblyopic eyes. Conclusion Strabismus amblyopia damage, nerve conduction time delay occurs mainly in the visual pathway of the retina above the site, the retina was not significantly impaired.