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BPPV是一种外周性迷路综合征,它是由一定的位置变化而引起的短暂、剧烈的眩晕发作。常伴有自主神经症状。患者在向患侧迷路倾倒时即诱发眩晕和眼震。眼震特点是,呈扭转性、有典型的潜伏期,发作短暂,不超过30秒、恢复坐位时眼震方向改变及呈可疲性。但近来发现在50％的BPPV病例中在悬头位时有继发性眼震出现。其发病机理认为可能是由耳石膜的耳石脱落沉积于后半规管(PSC)嵴帽所致。Schuknecht称之为嵴顶沉石症。近来在这种BPPV病例中发现其症状虽与BPPV相似,但与PSC嵴顶沉石症不尽相同。由此作者推测其病程可能起源于水平半规管(HSC)。
BPPV is a peripheral labyrinth syndrome, which is caused by a certain position of the short, dramatic vertigo. Often accompanied by autonomic symptoms. Patient dizziness and nystagmus when falling to the affected side. Nystagmus is characterized by torsion, a typical incubation period, episodes of short, no more than 30 seconds, to restore the nystagmus direction when sitting and was fatigued. However, it has recently been found that secondary nystagmus occurs at the dart position in 50% of BPPV cases. The pathogenesis that may be otolithic otolith deposited on the semicircular canal (PSC) crest cap caused. Schuknecht calls it crest stone. Recently in this case of BPPV found that although the symptoms are similar to BPPV, but with the top of the stone crest rock PSC vary. The authors speculate that its duration may originate in the level of semicircular canal (HSC).