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这三例,反映出矽肺病变进展过程的一般和特殊的某些侧面,也反映了X线与病理之间的相一致和存在差距的情况,使我们很受启发。为此,报告如下,以供参考。例一,男,53岁,隧道开挖工12年。1966年4月胸片发现为粗大弥漫分布矽结节,上中较密集,在此基础上融合呈蝶翼状,逐步向纵隔向肺门收缩。约7年后,与肺门影重叠,肺门增大,致密,向上收缩,右气管旁有淋巴节肿大影,病灶附近的气肿征明显,胸膜对称性加厚。心脏由狭长而逐步横径加大,肺动脉段凸出等肺心病征日益明显(图1见81页)。1974年6月死亡,尸检发现:双肺满布粗大矽结节,伴双中上多个矽肺块形成(左
These three cases, which reflect the general and specific aspects of the progression of silicosis, also reflect the agreement and the gap between the X-ray and the pathology and are very much inspired. For this reason, the report is as follows for reference. Example 1, male, 53 years old, tunnel excavation worker for 12 years. April 1966 chest X-ray findings were coarse diffuse distribution of nodules, the more dense, on the basis of the merger was a wing-shaped, and gradually to the hilar and contraction of the mediastinum. About 7 years later, overlap with the hilar shadow, hilar enlargement, dense, upward contraction, lymphadenopathy next to the right tracheal enlargement, obvious signs of emphysema near the lesion, pleural symmetry thickened. Heart from the narrow and gradually increased diameter, pulmonary artery protruding bulge and other pulmonary heart disease is increasingly evident (Figure 1, page 81). Death in June 1974, autopsy found: lung covered with thick nodules, with double silicosis on the formation of a double block (left