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文献中已排除了脾脏大小与门脉压力之间的相互关系,但已证实,存在一种引起脾脏和门脉系血流量增高的脾动脉高动力性成分.在因肝内梗阻、脾大、脾功能亢进及无曲张静脉出血的中度门脉高压患者中,脾切除可导致肝病的发展和影响以后门脉高压的外科治疗.因此,有人用脾动脉结扎或栓塞术以消除门脉高压中的脾脏高动力成分,以缩小脾脏,同时保存脾静脉流出量和改善周围循环的血细胞减少症.作者提出一种简便的技术,既可证实脾脏高动力成分的存在及其对门脉压的影响,同时可导致药物性脾切除,避免施行上述手术.本文对16例有周围血细胞减少症,明显髓样组织功能亢进和无出血的中度门脉高压患者(男7例,女9例,平均年龄34岁)进行了研究.6例经肝针吸活检证实为坏死后肝硬变,其余10例在门脉间隙中有纤维化及/或炎症.
The correlation between spleen size and portal pressure has been ruled out in the literature, but it has been shown that there is a high motility component of the splenic artery that causes increased blood flow to the spleen and portal system.As a result of intrahepatic obstruction, splenomegaly, Splenic hyperthyroidism and varicose vein hemorrhage in patients with moderate portal hypertension, splenectomy can lead to the development of liver disease and the impact of surgical treatment of portal hypertension.Therefore, some people with the splenic artery ligation or embolization to eliminate portal hypertension Splenic high motility component to reduce spleens while preserving splenic vein outflow and improve peripheral circulation of cytopenia The authors propose a simple technique that can both confirm the presence of high motility components of the spleen and its effect on portal pressure , At the same time can lead to drug-induced splenectomy, to avoid the above-mentioned operation.In this paper, 16 patients with peripheral cytopenia, marked myeloid hyperthyroidism and no bleeding moderate portal hypertension patients (7 males and 9 females, mean Age 34 years) were studied.6 cases of liver cirrhosis confirmed by liver needle aspiration biopsy, and the remaining 10 cases of fibrosis and / or inflammation in the portal space.