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内耳迷路由前、侧、后半规管,椭圆囊,球状囊组成。大脑皮层前庭代表区位于颞上后回上半部,颞顶交界处和岛叶中部。从内耳迷路经前庭神经,前庭神经核及脑干内侧纵束直至大脑皮层前庭代表区的整个通路称为前庭系。前庭神经系任何部位受损均可发生眩晕称前庭性眩晕。临床上必须详问病史及多方面检查才能获得确诊,将前庭性眩晕之定位及病因特点结合病例讨论如下: 一、因前庭周围感受器而引起之眩晕 [例1]女,44岁,住院号210388,1981年3月6日入院。患者自1968年以来阵发性眩晕、伴耳鸣,恶心、呕吐;每次发作持续数分钟至半天。每2～3个月发作一次,今年1月以来眩晕发作加剧右耳听力也渐减退。过去无中耳炎史。经检查:双耳鼓膜略内陷,未穿孔,电测听示右耳气导及骨导消失,眼震图示双耳前庭功能减退,以右侧者明显。X线
Inner ear lost before the road, side, after the semicircular canal, oval capsule, spherical capsule composition. The vestibular representation of the cerebral cortex is located in the upper part of the superior temporal gyrus, the junction of the temporal crest and the middle part of the insula. From the inner ear lost through the vestibular nerve, vestibular nucleus and medial brainstem longitudinal bundle until the cerebral cortical vestibular representation of the entire pathway known as the vestibular system. Any part of the vestibular nerve damage can occur vertigo called vestibular dizziness. The clinical history and multidimensional examination must be detailed to get the diagnosis. The location of the vestibular vertigo and the characteristics of the etiology are discussed as follows: First, dizziness caused by receptors in the vestibular space [Example 1] Female, 44 years old, hospitalization 210388 , March 6, 1981 admission. Patients have had paroxysmal vertigo since 1968 with tinnitus, nausea and vomiting; each episode lasts from minutes to half a day. Attacks occur every 2 to 3 months, and the onset of vertigo increases gradually from January to January this year. No past history of otitis media. After examination: a slight retraction of the eardrum, non-perforated, electrical measurements showed right auricle and bone conduction disappeared, nystagmus showed bilateral vestibular dysfunction, to the right obvious. X-ray