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氯胺酮Ketamine)和尼莫的平(Nimodipine)分别是NMDA受体和Ca~(2+)通道的拮抗剂。本文用免疫组化(ABC)方法在大鼠四管阻塞全脑缺血再循环模型上,观察了它们对全脑暂时性缺血诱导脑内C—fos癌基因蛋白(Fos)表达的翻转作用。结
Ketamine and Nimodipine are antagonists of NMDA receptors and Ca 2+ channels, respectively. In this study, we used immunohistochemistry (ABC) to observe the reversal effect of transient global cerebral ischemia on the expression of C-fos oncogene protein (Fos) in the rat model of four-tube occlusive global cerebral ischemia-reperfusion . Knot