论文部分内容阅读
肺心病心衰患者几乎都有心动过速,而且其心率在心衰得到初步控制后仍无明显下降,这不仅使患者有心慌不适的感觉,而且长时间的心动过速还增加心脏做功及耗氧量,不利于心脏功能的恢复。为克服这一不利因素.我们在1990年对经常规治疗心功能初步改善后心率仍>110次/分者给小量氨酰心安,观察到满意的效果后,于1991年～1992年在常规治疗的同时加用小剂量氨酰心安治疗36例,并选择同期住院章用常规治疗的36例做章盲对照,现将结果报告如下: 1 病例选择 氨酰心安组 男25例.女11例;年龄54～80岁,平均66岁;心衰病史1～6年,平均3.6年。常规治疗组;男23例,女13例;年龄53～79岁,平均65岁;心衰病史0.5～7年,平均3.5年。
Almost all heart failure patients with pulmonary heart disease have tachycardia, and the heart rate in the heart failure has not been significantly reduced after the initial control, which not only makes patients feel flustered, and prolonged tachycardia also increases cardiac work and consumption Oxygen is not conducive to the recovery of cardiac function. To overcome this unfavorable factor, we in 1990 after the initial improvement of conventional treatment of heart function heart rate is still> 110 beats / min to a small amount of atenolol, to observe the satisfactory results in 1991 to 1992 in the conventional At the same time with the treatment of small doses of atenolol treatment of 36 cases, and choose the same period of hospital treatment with conventional treatment of 36 cases of blind control, the results are reported as follows: 1 cases of atenolol male 25 cases. Female 11 cases ; Aged 54 to 80 years, mean 66 years; heart failure history of 1 to 6 years, an average of 3.6 years. Conventional treatment group; 23 males and 13 females; aged 53 to 79 years, mean 65 years; history of heart failure 0.5 to 7 years, an average of 3.5 years.