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缺血预适应的心脏保护与促进内源性活性物质释放有关 ,一氧化氮参与缺血预适应的早期与延迟保护作用的调节 ,其作用机制可能是通过cGMP PKC的途径开放ATP敏感的钾通道所介导。
Cardioprotection of ischemic preconditioning is related to the release of endogenous active substances. Nitric oxide is involved in the regulation of the early and delayed protection of ischemic preconditioning. The mechanism may be that ATP-sensitive potassium channels are opened through the cGMP PKC pathway Mediated.