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妊娠妇女易患肾盂肾炎,肾小球肾炎常因妊娠而恶化;妊娠中毒症以高血压、浮肿及蛋白尿为临床特征,与肾小球肾炎极相似。本文着重将妊娠期的肾脏病变与其相互之间的影响,作一综述。妊娠期的有关肾脏生理变化正常妊娠肾脏的长度约增加1 cm,同时伴有肾盏、肾盂及输尿管扩张。血液循环总量增加20～30%,以第30～36周为最显著;每分钟心排血量约增加30～40%,产后2～6周恢复正常。随着血液循环量及每分钟心排血量的增加,肾血流量及肾小球滤过率也增加30～50%。妊娠第16～20周,肾血流量增加
Pregnant women susceptible to pyelonephritis, glomerulonephritis often deteriorated due to pregnancy; gestosis to hypertension, edema and proteinuria as a clinical feature, very similar with glomerulonephritis. This article focuses on the pregnancy of kidney disease and its impact on each other, made a review. Physiological changes related to pregnancy during pregnancy Normal pregnancy, the length of the kidney increased by about 1 cm, accompanied by calyx, renal pelvis and ureteral dilatation. The total blood circulation increased by 20 to 30% to 30 to 36 weeks for the most significant; cardiac output per minute increased by about 30 to 40%, 2 to 6 weeks postpartum returned to normal. With the amount of blood circulation and cardiac output per minute increases, renal blood flow and glomerular filtration rate also increased by 30 to 50%. 16 to 20 weeks of pregnancy, renal blood flow increased