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Background: Bilateral simultaneous anterior ischemic optic neuropathy (AION) i s uncommon. We report a case of bilateral and nearly simultaneously occurring AI ON. Case: A 61-yearold man was referred to our hospital with bilateral optic di sc edema. Observations: Visual field testing demonstrated inferior nasal defect OD and inferior defect OS. Fluorescein angiography showed a delay of dye filling in the superior part of the optic disc in both eyes. The patient had poorly con trolled diabetes. A mild increase in erythrocyte sedimentation rates and c-reac tive protein was observed, but the results of temporal artery biopsy were negati ve. His optic discs were small and lacked biological cups, which has been identi fied as a risk factor for developing AION. Conclusions: The complications of the structural anomaly, also known as “disc at risk,”and diabetes might have caus ed the bilateral and nearly simultaneously occurringAION.
Background: Bilateral simultaneous anterior ischemic optic neuropathy (AION) is uncommon. We report a case of bilateral and almost simultaneously occurring AI ON. Case: A 61-yearold man was referred to our hospital with bilateral optic di sc edema. Observations: Visual field testing demonstrated inferior nasal defect OD and inferior defect OS. Fluorescein angiography showed a delay of dye filling in the superior part of the optic disc in both eyes. The patient had poorly con trolled diabetes. A mild increase in erythrocyte sedimentation rates and c-reac tive protein was observed, but the results of temporal artery biopsy were negati ve. His optic discs were small and lacked biological cups, which has been identi fied as a risk factor for developing AION. Conclusions: The complications of the structural anomaly, also known as “disc at risk,” and diabetes may have caus ed the bilateral and nearly simultaneously occurringAION.