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慢性心力衰竭(CHF)是以进行性左室收缩和(或)舒张功能不全为特征的临床综合征。持续性全身炎症、免疫反应激活可能是各种机制的最终致病环节。然而,关于肾素-血管紧张素-醛固酮(RAAS)系统激活、炎性细胞因子及自身免疫反应之间的内在联系却鲜有人提及。本研究对CHF患者在常规抗心衰治疗基础上加用阿托伐他汀,旨在探讨其对CHF患者RAAS系统与炎性因子的影
Chronic heart failure (CHF) is a clinical syndrome characterized by progressive left ventricular systolic and / or diastolic dysfunction. Sustained systemic inflammation, activation of immune response may be the ultimate pathogenic mechanisms of various mechanisms. However, little is known about the intrinsic link between the activation of the renin-angiotensin-aldosterone (RAAS) system, inflammatory cytokines and autoimmune responses. In this study, atorvastatin was added to CHF patients on the basis of conventional anti-heart failure treatment to explore the influence of RAAS system and inflammatory cytokines