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目的观察与分析胎膜早破对产妇产后感染及机体应激的影响情况。方法选取2015年6月-2016年11月该院收治的62例胎膜早破患者为观察组,同时期的62例非胎膜早破者为对照组,然后统计及比较两组的产后感染率,并比较观察组中不同胎膜早破类型患者的产后感染率,同时比较不同胎膜早破类型患者破膜后不同时间的应激激素及炎性应激指标水平。结果观察组的产后感染率明显高于对照组,而未足月胎膜早破者的产后感染率高于足月胎膜早破者,且未足月胎膜早破者破膜后不同时间的应激激素及炎性应激指标水平均高于足月胎膜早破者,均P<0.05,差异均有统计学意义。结论胎膜早破可导致产妇产后感染率升高,同时未足月胎膜早破的感染率相对更高,且机体应激程度也更为明显,因此应给予针对性的调节与干预。
Objective To observe and analyze the impact of premature rupture of membranes on maternal postpartum infection and body stress. Methods Sixty-two patients with premature rupture of membranes in our hospital from June 2015 to November 2016 were selected as observation group and 62 non-premature rupture of membranes in the same period as control group. Then, the postpartum infection Rates of postpartum infection in patients with different types of premature rupture of membranes in the observation group were compared. Stress hormones and inflammatory stress indicators at different times after rupture of membrane in different types of premature rupture of membranes were compared. Results The incidence of postpartum infection in the observation group was significantly higher than that in the control group, but the rate of postpartum infection in those who did not have full-term premature rupture of membranes was higher than those in full-term premature rupture of membranes, Of stress hormones and inflammatory stress indicators were higher than full-term premature rupture of membranes, both P <0.05, the differences were statistically significant. Conclusion Premature rupture of membranes can lead to the increase of postpartum infection rate, and the premature rupture of membranes is relatively high, and the degree of stress in the body is more obvious. Therefore, we should give targeted adjustment and intervention.