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Graves病(突眼性甲状腺肿)的眼病理不太了解,是否为一种内分泌抗体或是一种细胞介导的自动免疫的机制作为病理反应的基础尚未清楚,眼眶容量的增加继发了眶内压增加,导致视野缺损,视力下降,乳头水肿,眼肌麻痹,最后失明。双眼突出疼痛、结膜炎和球结膜水肿,眼睑回缩和眼外肌失调,引起很大不适。本组病人20例手术,其中17人为双侧,3人单侧。17例女性。病人年龄是25岁～72岁(平均44岁)。术前甲状腺机能均正常。X线片无急
The pathology of Graves’ disease is not well understood. Whether or not it is an endocrine antibody or a cell-mediated mechanism of autoimmunity has not been clearly established as a basis for the pathological response. The increase in orbital volume is secondary to the orbital Increased internal pressure, resulting in visual field defects, decreased vision, papillary edema, ophthalmoplegia, and finally blindness. Eyes prominent pain, conjunctivitis and conjunctival edema, eyelid retraction and extraocular muscle imbalance caused great discomfort. Twenty patients underwent surgery in this group, 17 of whom were bilateral and 3 unilateral. 17 cases of women. The patient’s age ranged from 25 to 72 years (mean 44 years). Preoperative thyroid function are normal. X-ray film is not urgent