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慢性应激诱发早期高血压，在高血压发生期（应激４～１６天），观察动脉压力感受反射敏感性变化。结果表明：动脉压力感受反射衰减是伴随血压升高而发生并与之平行发展，而与该时期交感紧张性升高的时程不相平行，压力感受反射心率效应的衰减早于肾交感神经活动抑制效应的衰减，压力感受传入机能在高血压发展后未见明显变化。应激过程血压未表现明显升高的大鼠，其动脉压力感受反射也未见明显变化。提示：伴随高血压发生过程的动脉压力感受反射衰减从属于中枢机能变化，它可能参与高血压的发展，导致该衰减的主要动因可能是异常血压本身传入信息，而不是交感紧张性升高。
Chronic stress induces early hypertension, and changes in arterial baroreflex sensitivity are observed during the onset of hypertension (stress 4 to 16 days). The results showed that the attenuation of arterial pressure-sensing reflex was accompanied by the rise of blood pressure and paralleled the rise of sympathetic tone during this period, but the attenuation of heart rate reflex was earlier than that of renal sympathetic nerve Inhibition of the effect of attenuation, pressure sensing in the introduction of function in the development of hypertension no significant change. The stress process did not show significant increase in blood pressure in rats, the arterial pressure sensory reflex no significant change. It is suggested that the attenuation of arterial baroreceptor reflex accompanying the process of hypertension is subordinate to the change of central nervous system. It may be involved in the development of hypertension. The main cause of this decay may be the abnormal blood pressure itself rather than the sympathetic tone.