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为了探讨交感神经对失血性休克过程中淋巴微循环变化的影响，应用显微电视录象技术，观察了切断内脏大神经的大鼠在失血性休克及输血、补液过程中肠系膜微淋巴管（ＭＬ）收缩性的变化。结果表明，去神经后ＭＬ自主收缩频率及总收缩活性指数（ＩｎｄｅｘⅡ）、淋巴管动力学指数（Ｌ．Ｄ－Ｉｎｄｅｘ）显著降低，失血性休克时神经完整组及去神经组ＭＬ的自主收缩性均降低，神经完整组在回输血液及输液期，ＭＬ自主收缩性显著高于休克前，而去神经组的ＭＬ收缩性仍维持低水平，且显著低于神经完整组（Ｐ＜０．０１）。输液后期，去神经组的血压显著低于神经完整组。结果提示，失血性休克时，ＭＬ收缩性降低，输血输液可提高ＭＬ收缩性及回升血压，休克逆转的这些变化可能是依靠交感神经的调控实现的。
In order to investigate the effect of sympathetic nerve on the changes of lymphatic microcirculation during hemorrhagic shock, we used microsurgical video recording technique to observe the effect of mesenteric micro-lymphatic vessels (ML Contractility changes. The results showed that the autonomic contractile frequency, the total systolic activity index (LII) and the L-D index of ML after denervation were significantly lower than those of the intact group and the denervated group (P <0.01). In contrast, the contractility of spontaneous contractility of ML group was significantly lower than that of shock group (P <0.01), while the contractility of ML group was still lower than that of intact nerve group ). Late infusion, denervated blood pressure was significantly lower than the complete group of nerves. The results suggest that, during hemorrhagic shock, ML contractility decreased, transfusion infusion can improve ML contractility and blood pressure, these changes in shock reversal may be achieved by sympathetic regulation.