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成人呼吸窘迫综合征(ARDS)的临床、放射学和病理学特征,尽管有各种各样急速引起的损害,但表明一些共同的效应器机制;详尽的证据显示与嗜中性细胞有关。嗜中性细胞从肺血管系统中分离并移入肺间质和肺泡间隙。实验动物的ARDS样病变可由以下情况引起:心肺转流、内毒素血症、出血性休克、肺炎球菌菌血症、血管内补体潘化、微血管形成或化学制剂,并且在上述这些情况下肺同样也
The clinical, radiological and pathological features of adult respiratory distress syndrome (ARDS), despite a variety of rapidly evolving lesions, suggest some common effector mechanisms; exhaustive evidence is associated with neutrophils. Neutrophils are separated from the pulmonary vasculature and migrate into the interstitium and alveolar spaces of the lungs. ARDS-like lesions in experimental animals can be caused by cardiopulmonary bypass, endotoxemia, hemorrhagic shock, pneumococcal bacteremia, intravascular complement pancreas, microvascular formation or chemical agents, and in the above cases the lungs are also and also