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本文测定了35例不稳定心绞痛(UA)、30例高血压心脏病(HHD)患者以及50例正常人的血浆抗凝血因子蛋白C(Pc)和纤维结合蛋白(Fn).结果表明:UA患者血浆Pc明显高于正常人和HHD患者(均P<0.001).HHD患者血浆Pc活性与正常人比较无显著性差异(P>0.05).UA患者和HHD患者血浆Fn明显低于正常人(P<0.001).但UA患者和HHD患者血浆Fn含量无显著性差异(P>0.05).提示冠心病UA时机体处于高凝状态,Pc活性增高是患病机体对这种高凝状态的反应性代偿.Fn参与了机体凝血机制的调节.
In this paper, we measured plasma anticoagulant protein C (Pc) and fibronectin (Fn) in 35 patients with unstable angina (UA), 30 hypertensive heart disease (HHD) and 50 healthy controls.The results showed that UA The plasma Pc of patients with HHD was significantly higher than that of normal subjects and HHD patients (all P <0.001) .Plasma activity in HHD patients was not significantly different from that in normal subjects (P> 0.05) P <0.001) .However, there was no significant difference in plasma Fn levels between UA patients and HHD patients (P> 0.05), suggesting that the body of the coronary heart disease is hypercoagulable in UA and the increased Pc activity is the response of the diseased body to this hypercoagulable state Sexual compensations .Fn involved in the regulation of the body’s coagulation mechanism.