论文部分内容阅读
阻塞性睡眠呼吸暂停低通气综合征(OSAHS)的主要特点是睡眠过程中由于上气道完全或部分阻塞导致呼吸暂停从而产生慢性间歇性低氧、二氧化碳储留,反复胸腔内负压增大、反复微觉醒、睡眠结构异常、白天嗜睡及记忆力下降,并可引起自主神经功能紊乱等。血氧反复降低,从而引起血压上升,正常昼夜血压节律消失。传统的高血压治疗主要依赖于药物,而OSAHS引起的高血压往往十分顽固,治疗效果差,多种药物联合应用也不能将血压控制在理想范围。治疗OS-AHS能有效控制继发于OSAHS的高血压
Obstructive sleep apnea-hypopnea syndrome (OSAHS) is characterized by sleep during the upper airway obstruction caused by complete or partial obstruction to produce intermittent intermittent hypoxemia, carbon dioxide storage, increased intrathoracic negative pressure, Repeatedly arousal, abnormal sleep structure, daytime drowsiness and memory loss, and can cause autonomic dysfunction. Blood oxygen repeatedly decreased, causing the blood pressure to rise, the normal diurnal blood pressure rhythm disappears. The traditional treatment of hypertension mainly depends on the drug, and OSAHS-induced hypertension is often very stubborn, the treatment effect is poor, a variety of drugs can not be combined with the application of blood pressure control in the ideal range. OS-AHS treatment can effectively control hypertension secondary to OSAHS