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本文用分别补充VE(100ppm)的低硒(0.011ppm)和加硒(0.084ppm)饲料饲养并繁殖大鼠。血硒测定表明,母代和子代的低硒组均明显地低于母代和子代的加硒组(p<0.01);新生子代低硒鼠的骨骼肌硒含量亦显著地低于新生子代加硒鼠。但是,组织学上未检见实验大鼠骨骼肌的病变。组织化学上,新生子代鼠的骨骼肌纤维对Nitro BT染色不呈现A、Ⅰ和An型骨骼肌纤维的分型。母代和子代低硒大鼠骨骼肌的A型和Ⅰ型肌纤维的数量及A/A+Ⅰ比值同母代和子代的加硒组相比无明显差异(p>0.05)。结果表明.在补充VE的低硒情况下,大鼠骨骼肌琥珀酸脱氢酶的活性不受硒缺乏的影响。
Rats were bred and bred with low selenium (0.011 ppm) and selenium-added (0.084 ppm) feed supplemented with VE (100 ppm), respectively. Blood selenium measurements showed that the selenium content of skeletal muscle of newborn offspring selenium mice was also significantly lower than that of the newborn (P <0.01) On behalf of selenium mice. However, histological examination of skeletal muscle lesions in experimental rats was not observed. Histochemically, skeletal muscle fibers of newborn offspring did not exhibit A, I and An skeletal muscle fiber types for Nitro BT staining. The numbers of A type and type I muscle fibers and the ratio of A / A + Ⅰ in the skeletal muscle of the selenium and offspring of the mother and offspring did not show significant difference (p> 0.05) compared with those of the selenium group. The results showed that in the case of low selenium supplemented with VE, the activity of succinate dehydrogenase in skeletal muscle of rats was not affected by selenium deficiency.