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疑窦在心律失常中,绝大部分早搏、阵发性心动过速、房颤、房朴、反复心律,甚至心室颤动,都可能与折返激动有关。对折返激动的形成与药物消除折返的机制,笔者在查阅有关资料的过程中,发现一些疑窦,有必要提出进一步探讨。首先,病损区是否即单向阻滞区的问题,有的回答是肯定的;也有人认为,单向阻滞区还可以是未受损的部分。其次,心肌细胞受损区的有效不应期问题。局部病变或缺血,心室的部分浦氏纤维末端的有效不应期缩短;而另一些专著认为,心肌在缺血情况下,膜电位降低,快钠通道失活,使浦氏纤维等快反应电位变为慢反应电
In sinus arrhythmias, most of the premature beat, paroxysmal tachycardia, atrial fibrillation, atrial fibrillation, repeated heart rhythm, and even ventricular fibrillation may be related to reentry stimulation. On the formation of rejuvenation and drug elimination reentry mechanism, the author in the process of access to relevant information, found some doubts, it is necessary to propose further study. First of all, whether the lesion area is a one-way block area or not, some of the answers are affirmative; others think that the unidirectional block area can also be an intact part. Second, the effective refractory period myocardial cell damage problems. Local lesions or ischemia, the ventricular part of the Purification of the effective shortening of the fiber end shortened; while others monograph that the myocardial ischemia in the case of membrane potential decreased fast sodium inactivation of the fiber so fast response Potential becomes slow reactive