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肝脏对脂蛋白的合成、代谢及运转起枢纽作用,肝硬化患者肝脏细胞功能低下,脂蛋白、载脂蛋白的合成,血浆脂蛋白代谢转化、降解均发生障碍而导致脂蛋白质和量异常,引起血清胆固醇酯和高密度脂蛋白含量下降。脂蛋白(α)[Lp(α)]作为心脑血管疾病的独立危险因素已被国内外确认,而Lp(α)在肝硬化患者的变化很少报道,我们对30例肝硬化患者进行血清Lp(α)的定量测定,并探讨其临床意义。 一、对象和方法 肝硬化组共检测30例,均来自住院病例,其中肝硬化代偿期8例,失代偿期22例;男性22例,女性8例;年龄为28～70岁,平均年龄50.2岁。对照
The liver plays a pivotal role in the synthesis, metabolism, and functioning of lipoproteins, with low liver cell function, synthesis of lipoproteins, apolipoproteins, metabolic conversion of plasma lipoproteins, and degradation of hepatic cirrhosis, resulting in dyslipoproteinemia and anomalies that cause Serum cholesterol ester and high density lipoprotein levels decreased. As an independent risk factor for cardiovascular and cerebrovascular diseases, lipoprotein (α) [Lp (α)] has been confirmed both at home and abroad. However, changes in Lp (α) in patients with cirrhosis are rarely reported. Seventy patients with cirrhosis Lp (α) quantitative determination, and explore its clinical significance. First, the object and methods of liver cirrhosis were detected in 30 cases, all from in-patient cases, including cirrhosis of the decompensation in 8 cases, 22 cases of decompensation; 22 males and 8 females; aged 28 to 70 years, mean Age 50.2 years old. Control