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作者观察了130例脑血管疾患者,其中一时性供血不足62例,脑梗塞68例,同时以80名正常人为对照。观察项目包括自发性血小板聚集,ADP诱发的血小板聚集以及一些药物对血小板聚集的影响。结果发现将患者富有血小板的血浆在37℃每分钟300转速下搅拌15分钟,有50例患者有自发性血小板聚集,其中21例为一时性供血不足。正常人无自发性血小板聚集。有自发性血小板聚集的富有血小板的血浆对ADP也敏感。ADP最终浓度为0.25微克分子时即可诱发血小板聚集(正常值为0.75±0.2微克分子)。将有自发性血小板聚集的富有血小板的血浆与EDTA(15毫克分子)、腺苷(0.1毫克分子)或
The authors observed 130 cases of cerebrovascular disease, including transient hypoperfusion in 62 cases, 68 cases of cerebral infarction, while 80 normal controls. Observations included spontaneous platelet aggregation, ADP-induced platelet aggregation, and the effects of some drugs on platelet aggregation. The results showed that patients with platelet-rich plasma at 37 ℃ 300 rpm stirring for 15 minutes, 50 patients had spontaneous platelet aggregation, of which 21 cases of transient hypoperfusion. Normal spontaneous platelet aggregation. Platelet-rich plasma with spontaneous platelet aggregation is also sensitive to ADP. ADP induces platelet aggregation at a final concentration of 0.25 micromolar (normal 0.75 ± 0.2 micromolar). Platelet-rich plasma with spontaneous platelet aggregation was incubated with EDTA (15 milligram), adenosine (0.1 milligram) or