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骨髓型急性放射病 (ＡＲＳ)在急性期、恢复期及照射后 6年期均显示红细胞膜阴离子交换蛋白 (Ａｎｉｏｎｅｘｃｈａｎｇｅｒ,ＡＥ ;ｂａｎｄ 3ｐｒｏｔｅｉｎ ,带 3蛋白 )功能异常 ,以新生红细胞病变严重 ,阴离子转运速率明显延迟 ,对阴离子通透特异抑制剂ＤＩＤＳ抑制效应下降
In the acute stage, convalescence stage and 6 years after irradiation, the myeloid acute radiation sickness (ARS) showed the abnormal function of Anion exchanger (AE; band 3 protein) with erythrocyte disease. The rate of anion transport Obvious delay, decreased the inhibitory effect of DIDS, a specific inhibitor of anion exchange