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冠状动脉痉挛作为变异型心绞痛的原因已为人接受,并在有S—T段压低的心绞痛中令人信服地得到了证明。当死后解剖发现几乎所有的心绞痛患者均有严重的冠状动脉粥样硬化性狭窄后,一百多年前所假定的冠状动脉痉挛引起心绞痛的机制曾被否定。对冠状动脉痉挛认识的复兴,从两方面影响我们对心绞痛的探讨。直接地关系到治疗;而间接地则有助于动摇长达数十年的认识障碍,即心绞痛唯一应受重视的原因是,在有器质性冠状动脉狭窄存在的情况下,心脏需氧量的过度增加。
Coronary artery spasms have been accepted as a cause of variant angina and have been convincingly demonstrated in angina pectoris with depression in the S-T segment. After death, the mechanism of angina pectoris that had been hypothesized more than one hundred years ago was negated once autopsy revealed that almost all angina patients had severe coronary atherosclerotic stenosis. The revival of awareness of coronary artery spasm affects our discussion of angina in two ways. Directly related to treatment; Indirectly help to shake up decades of cognitive impairment, angina pectoris the only reason why attention should be given in the presence of organic coronary artery stenosis, cardiac oxygen demand Excessive increase.